Hyperventilation syndrome is the initial stage of the deep breathing disease. The theory is based on up-to-date concepts of the grandiose biological role of CO2 in providing health of humanity and fauna, as well as on physiological mechanisms of CO2 effects on the organism, all its systems, flora and fauna.
Carbon dioxide is the staple for all the living matter on the Earth. Plants take it from the air, animals eat plants, and people eat both. Huge percentages of CO2 in the air of ancient times have come down to our minuscule 0.03%. Absorption of this residue by plants may inevitably lead to end of life on Earth. I reported that at the World Geochemistry Congress in Moscow in 1972.
Metabolism in human and animal cells developed in ancient geological epochs when concentration of carbon dioxide in air and water was dozens per cent. Therefore, a specific cellular concentration of CO2 is a part and parcel of normal biochemical processes. In the course of evolution the human organism and the highest animals have developed a self-governing aerial system in the form of pulmonary alveolar air which contains about 6.5% of CO2 and 7% less oxygen than in the ambient air. This is apparently the minimum level of CO2 that provides normal metabolic activities in cells.
For example, reduction of CO2 in the lungs due to hyperventilation offsets рН to the alkaline medium which alters vitamin and ferment activity. When the activity of metabolic regulators changes, normal metabolism shutters and this leads to loss and death of cells.
If CO2 comes down to 3% and рН offsets to 8%, the organism dies. Destructive effects of hyperventilation via creating CO2 deficiency in the organism have been verified by numerous experiments, first started by the famous physiologist D. Henderson in 1909. Henderson connected animals to a hyperventilation machine and they died.
Evolution has worked out the following protective mechanisms to stabilize CO2 in the lungs:
а) bronchospasm and vasospasm;
b) increased production of cholesterol by the liver: it works as a biological insulation that consolidates cell membranes in the lungs and vessels; and
c) lower blood pressure (hypotension), which reduces loss of CO2.
However, bronchospasm and vasospasm constrict oxygenation of the brain, kidney and cells of other organs. Diminution of CO2 in the blood enhances reactogenicity of oxygen and hemoglobin to downgrade oxygenation of cells (the Verigo-Bohr effect). Reduced oxygenation of tissues results in hypoxia. On reaching hazardous levels, hypoxia may cause higher blood pressure (hypertension) in some individuals. High blood pressure increases the bloodstream through constricted vessels to enhance oxygenation of the vitally important cells. Tissue hypoxia shrinks the level of oxygen in the venous blood which then brings about varicose veins in legs and develops varix, or, alternately, varicose hemorrhoidal veins with consequent hemorrhoids.
Gradual subtraction of CO2 from the blood boosts blood coagulation, and combined with deceleration of bloodstream in the veins it may cause thrombophlebitis. Acute hypoxia of the vital organs irritates the respiratory center creating the dominant activation there. This deepens breath, produces the feel of dyspnea (or air shortage for deep breathers) and locks the vicious circle (the positive feedback that persistently intensifies or deepens breath, stimulating the above disorders-illnesses).
Removal of CO2 from the nervous cells decreases their excitability threshold, agitates the entire nervous system and leads to irritability, insomnia, extreme nervous tension, unfounded suspiciousness, fear, or even fainting and epileptic seizure. Simultaneously, the respiratory center grows more and more agitated. That is how the second vicious circle of nervous excitement circulation locks. If metabolism is disturbed and the nervous cells suffer from hypoxia, the nervous system becomes exceptionally sensitive to external stimuli and stresses. This is why CO2 deficiency caused, namely, by hyperventilation affects the nervous system firsthand.
Symptoms of various disorder combinations in deep breathers are incredibly versatile. The traditional disease analysis has brought us to giving various deep breathing disease symptoms (such as bronchospasm, cardiac vasospasm, high or low blood pressure, or syncope with spasms) names of totally different diseases: bronchial asthma, stenocardia, high blood pressure, and epilepsy. The latter cause side-effects: pneumoscelorsis, vasosclerosis, cardiac infarction, and stroke. All of these are the main elements of early senility, decrepitude, disablement and, finally, death.
The above physiological laws explain the malignant (venomous) effects of deep breathing and give grounds for the only scientific principle of eliminating disorders (called diseases), i.e. by increasing the level of CO2 in the body. This is the principle we have based on our method of the voluntary hyperventilation elimination (VHE), or voluntary breath normalization (VBN).
If the breathing depth drops below normal and the level of CO2 in the organism grows 0.5% above, there will be no negative symptoms. Quite on the contrary, ex-patients with deep breathing (i.e. bronchial asthma, stenocardia, or high blood pressure) develop symptoms of un-endurance, which has been consistently observed for almost two decades. We found out that extreme de-deepening of breath does not end in harmful aftereffects. This is how we have actually discovered the main law of death: the deeper you breathe the stronger the illness is, and the closer death draws. Reversely, the shallower the breath is, the healthier, stronger and more durable the organism.
The deep breathing disease theory is given out in my lecture, “The Discovery of Deep Breath Being the Main Cause of Allergy, Sclerosis, Psychosis, Tuberculosis, Pre-cancer and Other Western Civilization Degeneration, Degradation, and Ailment Symptoms and Death”.